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Disease Info Card
Aseptic Peritonitis
Information about Aseptic Peritonitis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Aseptic Peritonitis
Most recent studies have shown that Aseptic Peritonitis shares some biological mechanisms with abdominal-pain, arthritis, bacterial-infections, bacterial-peritonitis, brucellosis, diabetes-mellitus, exanthema, familial-mediterranean-fever, inflammation, inflammatory-response, kidney-failure-chronic, leukocytosis, malignant-neoplasms, myalgia, neoplasms, pain, peritonitis, tissue-adhesions, vomiting.
Among the many pathways, these few ones have gauged particular interests from scientists studying Aseptic Peritonitis, and have been seen in publications frequently: Acute Inflammatory Response, Apoptotic Cell Clearance, Cell Activation, Cell Adhesion, Cell Migration, Chemokine Production, Chemotaxis, Cytokine Production, Diapedesis, Diuresis, Hypersensitivity, Immune Response, Inflammatory Response, Lymphangiogenesis, Neutrophil Chemotaxis, Neutrophil Extravasation, Phagocytosis, Sensitization, T Cell Activation, Transport
Quite a number of genes have been found to play important roles in Aseptic Peritonitis, such as ALB, BRCA1, CCL5, CRP, CTSC, CTSG, CXCL2, ELANE, IL10, IL6, ITGAM, ITGB2, LTBR, MSR1, PRTN3, TNF, TNFRSF1A, VEGFC. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.